Rabbit anti Mouse Albumin antibody recognizes mouse albumin, a serum hepatic soluble protein which constitutes approximately 50% of blood serum protein, acting as a regulator of blood colloidal osmotic pressure and hence blood volume. Albumin is also an important transporter of substances within the bloodstream, such as fatty acids, thyroid hormones, metal ions and steroids.
Rabbit anti Mouse Albumin antibody (AHP1478) may cross-react with albumin from other species.
- Target Species
- Product Form
- Serum - liquid
- Antiserum Preparation
- Antiserum to mouse albumin were raised by repeated immunisation of rabbits with highly purified antigen.
- Preservative Stabilisers
- Store at +4oC. DO NOT FREEZE.
This product should be stored undiluted. Should this product contain a precipitate we recommend microcentrifugation before use.
- 12 months from date of despatch
- Entrez Gene
- GO Terms
cellular response to starvation
hemolysis by symbiont of host erythrocytes
pyridoxal phosphate binding
negative regulation of apoptosis
maintenance of mitochondrion location
- For research purposes only
This product has been reported to work in the following applications. This information is derived from testing within our laboratories, peer-reviewed publications or personal communications from the originators. Please refer to references indicated for further information. For general protocol recommendations, please visit the antibody protocols page.
Applications of Albumin antibody
Where this product has not been tested for use in a particular technique this does not necessarily exclude its use in such procedures. Suggested working dilutions are given as a guide only. It is recommended that the user titrates the product for use in their own system using the appropriate negative/positive controls.
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Secondary Antibodies Available
Useful Reagents Available
Application Based External Images
Product Specific References
References for Albumin antibody
Kovtunovych, G. et al (2010) Dysfunction of the heme recycling system in heme oxygenase 1-deficient mice: effects on macrophage viability and tissue iron distribution.
Blood. 116: 6054-62.
Ruseva, M.M. et al. (2013) Loss of properdin exacerbates C3 glomerulopathy resulting from factor H deficiency.
J Am Soc Nephrol. 24 (1): 43-52.
Vernon, K.A. et al. (2016) Partial Complement Factor H Deficiency Associates with C3 Glomerulopathy and Thrombotic Microangiopathy.
J Am Soc Nephrol. 27 (5): 1334-42.
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